Robocop

yes avec lazed c'est ce qu'il faudrait faire

Matin : 4 oeuf + 2 bananes + 50gr d'avoine 10h : 30g de prot + 15g d'amandes 12h30 : 50g de riz complet + 200g de poulet + légumes + huile de colza 16h : 100g de poulet + 50gr de riz Intraînement : 15g de BCAA + 50 g de maltodextrine Post training : 50g de whey + 50gr d'avoine Soir : 200g de poulet + légumes + huile de colza j'aurai plus vu comme ça, même quantité de glucides mais placés différement

ta cure c'est tbol tu reste sur ton tbol.

j'aime pas comment sont placés les glucides

ya pas de copie ya juste le lien ensuite l'autre texte vient d'ailleurs je suis en règle !

tu vas voir la congestion avec le turi..

https://thinksteroids.com/articles/why-use-both-clomid-and-nolvadex-together-for-pct/ la il parle du fait que le nolvadex agit aussi bien que le clomid a un dosage moindre, donc sa serait surtout un moyen de diminuer les effets négatifs de l'un et l'autre en les combinant pour réduire le dosage en sorte. la un petit texte clomid vs nolva : "Nolvadex vs Clomid" by William Llewellyn I have received a lot of heat lately about my preference for Nolvadex over Clomid, which I hold for all purposes of use (in the bodybuilding world anyway); as an anti-estrogen, an HDL (good) cholesterol-supporting drug, and as a testosterone-stimulating compound. Most people use Nolvadex to combatgynecomastia over Clomid anyway, so that is an easy sell. And for cholesterol, well, most bodybuilders unfortunately pay little attention to this important issue, so by way of disinterest, another easy opinion to discuss. But when it comes to using Nolvadex for increasing endogenous testosterone release, bodybuilders just do not want to hear it. They only seem to want Clomid. I can only guess that this is based on a long rooted misunderstanding of the actions of the two drugs. In this article I would therefore like to discuss the specifics for these two agents, and explain clearly the usefulness of Nolvadex for the specific purpose of increasing testosterone production. Clomid and Nolvadex I am not sure how Clomid and Nolvadex became so separated in the minds of bodybuilders. They certainly should not be. Clomid and Nolvadex are both anti-estrogens belonging to the same group of triphenylethylene compounds. They are structurally related and specifically classified as selective estrogen receptor modulators (SERMs) with mixed agonistic and antagonistic properties. This means that in certain tissues they can block the effects of estrogen, by altering the binding capacity of the receptor, while in others they can act as actual estrogens, activating the receptor. In men, both of these drugs act as anti-estrogens in their capacity to oppose the negative feedback of estrogens on the hypothalamus and stimulate the heightened release of GnRH (Gonadotropin Releasing Hormone). lh - leutenizing hormone - output by the pituitary will be increased as a result, which in turn can increase the level of testosterone by the testes. Both drugs do this, but for some reason bodybuilders persist in thinking that Clomid is the only drug good at stimulating testosterone. What you will find with a little investigation however is that not only is Nolvadex useful for the same purpose, it should actually be the preferred agent of the two. Pituitary Sensitivity to GnRH Studies conducted in the late 1970's at the University of Ghent in Belgium make clear the advantages of using Nolvadex instead of Clomid for increasing testosterone levels (1). Here, researchers looked the effects of Nolvadex and Clomid on the endocrine profiles of normal men, as well as those suffering from low sperm counts (oligospermia). For our purposes, the results of these drugs on hormonally normal men are obviously the most relevant. What was found, just in the early parts of the study, was quite enlightening. Nolvadex, used for 10 days at a dosage of 20mg daily, increased serum testosterone levels to 142% of baseline, which was on par with the effect of 150mg of Clomid daily for the same duration (the testosterone increase was slightly, but not significantly, better for Clomid). We must remember though that this is the effect of three 50mg tablets of Clomid. With the price of both a 50mg Clomid and 20mg Nolvadex typically very similar, we are already seeing a cost vs. results discrepancy forming that strongly favors the Nolvadex side. But something more interesting is happening. Researchers were also conducting GnRH stimulation tests before and after various points of treatment with Nolvadex and Clomid, and the two drugs had markedly different results. These tests involved infusing patients with 100mcg of GnRH and measuring the output of pituitary lh - leutenizing hormone - in response. The focus of this test is to see how sensitive the pituitary is to Gonadotropin Releasing Hormone. The more sensitive the pituitary, the more lh - leutenizing hormone - will be released. The tests showed that after ten days of treatment with Nolvadex, pituitary sensitivity to GnRH increased slightly compared to pre-treated values. This is contrast to 10 days of treatment with 150mg Clomid, which was shown to consistently DECREASE pituitary sensitivity to GnRH (more lh - leutenizing hormone - was released before treatment). As the study with Nolvadex progresses to 6 weeks, pituitary sensitivity to GnRH was significantly higher than pre-treated or 10-day levels. At this point the same 20mg dosage was also raising testosterone and lh - leutenizing hormone - levels to an average of 183% and 172% of base values, respectively, which again is measurably higher than what was noted 10 days into therapy. Within 10 days of treatment Clomid is already exerting an effect that is causing the pituitary to become slightly desensitized to GnRH, while prolonged use of Nolvadex serves only to increase pituitary sensitivity to this hormone. That is not to say Clomid won't increase testosterone if taken for the same 6 week time period. Quite the opposite is true. But we are, however, noticing an advantage in Nolvadex. The Estrogen Clomid The above discrepancies are likely explained by differences in the estrogenic nature of the two compounds. The researchers' clearly support this theory when commenting in their paper, "The difference in response might be attributable to the weak intrinsic estrogenic effect of Clomid, which in this study manifested itself by an increase in transcortin and testosterone/estradiol-binding globulin [sex hormone binding globulin ] levels; this increase was not observed aftertamoxifen treatment". In reviewing other theories later in the paper, such as interference by increased androgen or estrogen levels, they persist in noting that increases in these hormones were similar with both drug treatments, and state that," ?a role of the intrinsic estrogenic activity of Clomid which is practically absent in Tamoxifen seems the most probable explanation". Although these two are related anti-estrogens, they appear to act very differently at different sites of action. Nolvadex seems to be strongly anti-estrogenic at both the hypothalamus and pituitary, which is in contrast to Clomid, which although a strong anti-estrogen at the hypothalamus, seems to exhibit weak estrogenic activity at the pituitary. To find further support for this we can look at an in-vitro animal study published in the American Journal of Physiology in February 1981 (2). This paper looks at the effects of Clomid and Nolvadex on the GnRH stimulated release of lh - leutenizing hormone - from cultured rat pituitary cells. In this paper, it was noted that incubating cells with Clomid had a direct estrogenic effect on cultured pituitary cell sensitivity, exerting a weaker but still significant effect compared to estradiol. Nolvadex on the other hand did not have any significant effect on lh - leutenizing hormone - response. Furthermore it mildly blocked the effects of estrogen when both were incubated in the same culture. Conclusion To summarize the above research succinctly, Nolvadex is the more purely anti-estrogenic of the two drugs, at least where the hpta - hypothalamic-pituitary-testicular axis - (Hypothalamic-Pituitary-Testicular Axis) is concerned. This fact enables Nolvadex to offer the male bodybuilder certain advantages over Clomid. This is especially true at times when we are looking to restore a balanced hpta - hypothalamic-pituitary-testicular axis - , and would not want to desensitize the pituitary to GnRH. This could perhaps slow recovery to some extent, as the pituitary would require higher amounts of hypothalamic GnRH in the presence of Clomid in order to get the same level of lh - leutenizing hormone - stimulation. Nolvadex also seems preferred from long-term use, for those who find anti-estrogens effective enough at raising testosterone levels to warrant using as anabolics. Here Nolvadex would seem to provide a better and more stable increase in testosterone levels, and likely will offer a similar or greater effect than Clomid for considerably less money. The potential rise in sex hormone binding globulin levels with Clomid, supported by other research (3), is also cause for concern, as this might work to allow for comparably less free active testosterone compared to Nolvadex as well. Ultimately both drugs are effective anti-estrogens for the prevention of gynecomastia and elevation of endogenous testosterone. au final si tu veux en utiliser que un tu as fais le mauvais choix

j'adore !

parce que y'a du jus de betterave !!

Superbe évolution tu as fais sa naturel ? tu manque de trapèzes par contre

n'y moi

En plus Kevol comme tu dis mais la il est bien en proteine il est pas en cure mais en cure sa peut etre une piste pour augmenter le taux de proteines

juste le dernier repas ou j'aurai pas mis de glucides a la place je les aurai mis post training (d'ailleurs tu as écris pré mais c'est post) pour moitié et l'autre moitié a 16h sinon diet cohérente pour moi mais il nous manques les totaux comme souvent.. et petit plus j'aurai mis un peu de zinc et de magnesium

veuve on voit qu"il est en cure lol

Toujours aussi horrible lol 1/ mauvais choix de produit comme dit jonas oriente toi vers l'anavar ou peut etre le tbol, ces cures paraissent facilent car il suffit d'avaler des cachets mais au final peu de gains et surtout perte de temps 2/ tamoxifene (nolvadex) c'est un anti oestro ok mais on l'utilise pas en cure mais en relance, en cure on utilise l'aromasin ou l'arimidex 3/ proviron inutile dans ton cas 4/ liv52 la c'est bon ! 5/ il y a tout sur le forum pour les PCT lis

je dépasserai pas les 6 semaines, moi même je sors de 4s de win a 45mg j'ai sentis les reins je pissais bien orange et sa me faisait comme une infection urinaire sa piquait quand je pissais.. depuis l'arret l'urine c'est bien éclaircit

Le win pendant 8s je serai toi j'éviterai.. que ce soit en cachet ou en inj

Lorenzo becker.. merde sa fait chier pour lui

crampe au coeur je pense pas lol ou alors va voir un cardiologue

ben sa va fausser le résultat de l'analyse que l'endo te prescrira si il t'en prescrit une.. mais à mon avis il va pas servir a grand chose donc je pense que tu as bien fais de commencer. Les dosages sont bien pour moi, repose toi bien fais pas des entraînements trop long, manges bien (riche en lipides,omega 3)

pourquoi tu fais pas un peu plus durer la cure ? tu montes haut en dosage en plus 8 semaines de win... alors que tu pourrais faire un peu plus leger sur 12s pour construire un peux mieux

60mg de turi sa commence a faire beaucoup..

mixer clenbu et albu sa sert a rien c'est soit l'un soit l'autre et je te conseil l'albu ! Pour moi bien plus agréable

Tu as fais quoi comme cure avant ca ??

moi je le bois